Disease Info Card

Arrhythmogenic Right Ventricular Dysplasia

Information about Arrhythmogenic Right Ventricular Dysplasia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Arrhythmogenic Right Ventricular Dysplasia

Most recent studies have shown that Arrhythmogenic Right Ventricular Dysplasia shares some biological mechanisms with bundle-branch-block, cardiac-arrhythmia, cardiac-death, cardiac-fibrillation, cardiomyopathies, cardiomyopathy-dilated, dysplasia, fibrosis, heart-diseases, heart-failure, hypertrophic-cardiomyopathy, hypertrophy, left-bundle-branch-block, myocarditis, sudden-cardiac-death, sudden-death, syncope, tachycardia-ventricular, ventricular-dysfunction-right, ventricular-fibrillation.

Among the many pathways, these few ones have gauged particular interests from scientists studying Arrhythmogenic Right Ventricular Dysplasia, and have been seen in publications frequently: Aging, Cardiac Conduction, Cell Adhesion, Cell Death, Cell Migration, Cell Proliferation, Cell-cell Adhesion, Glomerular Filtration, Heart Development, Hyperphosphorylation, Innervation, Localization, Muscle Contraction, Organ Development, Pathogenesis, Programmed Cell Death, Swimming, Transdifferentiation, Translation, Transposition

Quite a number of genes have been found to play important roles in Arrhythmogenic Right Ventricular Dysplasia, such as DES, DSC2, DSC3, DSG2, DSP, DSPP, EMB, GJA1, IGKV1-5, JUP, PKP2, RYR2, SCN5A, TGFB3, TMEM43, UBN1, XPO1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.